“Inhibition of γ-Secretase Leads to an Increase in Presenilin-1” has been published in Molecular Neurobiology. This work was supported in part by JPND through the BIOMARKAPD project, selected in the 2011 Biomarkers call.
Category Archives: JPND Publications
“Another piece in the progranulin puzzle: special binding between progranulin and prosaposin creates additional lysosomal access” has been published in the Journal of Neurochemistry. This work was supported in part by JPND through the STRENGTH and RiMod-FTD projects, selected in the 2012 Risk Factors call.
“Perspectives of policy and political decision makers on access to formal dementia care: expert interviews in eight European countries” has been published in the journal BMC Health Services Research. This work was supported in part by JPND through the ActifCare project, selected in the 2012 Healthcare Evaluation call.
“Single-cell RNA sequencing: Technical advancements and biological applications” has been published in Molecular Aspects of Medecine. This work was supported in part by JPND through the DAMNDPATHS project, selected in the 2013 cross-disease analysis call.
“Improved method for cannula fixation for long-term intracerebral brain infusion” has been published in the Journal of Neuroscience Methods. This work was supported in part by JPND through the NeuroGEM project, selected in the 2013 cross-disease analysis call, and the PROP-AD project, selected in the 2015 JPco-fuND call.
“Sigma 1 receptor activation modifies intracellular calcium exchange in the G93AhSOD1 ALS model” has been published in the journal Neuroscience. This work was supported in part by JPND through the SOPHIA project, selected in the 2011 Biomarkers call.
“Polygenic Risk Score Analysis of Pathologically Confirmed Alzheimer’s Disease” has been published in the journal Annals of Neurology. This work was supported in part by JPND through the PERADES project, selected in the 2012 Risk Factors call.
“α-Synuclein transfer between neurons and astrocytes indicates that astrocytes play a role in degradation rather than in spreading” has been published in Acta Neuropathologica. This work was supported in part by JPND through the NeuTARGETS project, selected in the 2013 Cross-Disease Analysis call.
“KMT2A and KMT2B Mediate Memory Function by Affecting Distinct Genomic Regions” has been published in Cell Reports. This work was supported in part by JPND through the RiMod-FTD project, selected in the 2012 Risk Factors call.
“The role of de novo mutations in the development of amyotrophic lateral sclerosis” has been published in Human Mutation. This work was supported in part by JPND through the STRENGTH project, selected in the 2012 Risk Factors call, and the SOPHIA project, selected in the 2011 Biomarkers call.